High-mobility group box protein 1 (HMGB1): an alarmin mediating the pathogenesis of rheumatic disease.

Pisetsky DS, Erlandsson-Harris H, Andersson U.

Arthritis Res Ther. 2008;10(3):209. Epub 2008 Jun 30.

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Convergence and amplification of toll-like receptor (TLR) and receptor for advanced glycation end products (RAGE) signaling pathways via high mobility group B1 (HMGB1).

van Beijnum JR, Buurman WA, Griffioen AW.

Angiogenesis. 2008;11(1):91-9. Epub 2008 Feb 9. Review.

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High mobility group box 1 protein interacts with multiple Toll-like receptors.

Park JS, Gamboni-Robertson F, He Q, Svetkauskaite D, Kim JY, Strassheim D, Sohn JW, Yamada S, Maruyama I, Banerjee A, Ishizaka A, Abraham E.

Am J Physiol Cell Physiol. 2006 Mar;290(3):C917-24. Epub 2005 Nov 2.

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High mobility group box 1 in the pathogenesis of inflammatory and autoimmune diseases.

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HMGB1: endogenous danger signaling.

Klune JR, Dhupar R, Cardinal J, Billiar TR, Tsung A.

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Mechanisms of Disease: the role of high-mobility group protein 1 in the pathogenesis of inflammatory arthritis.

Jiang W, Pisetsky DS.

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Is HMGB1 an osteocyte alarmin?

Bidwell JP, Yang J, Robling AG.

J Cell Biochem. 2008 Apr 15;103(6):1671-80. Review.

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Inhibition of tumor angiogenesis by HMGB1 A box peptide.

Zhang CL, Shu MG, Qi HW, Li LW.

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The role of cell death in the pathogenesis of autoimmune disease: HMGB1 and microparticles as intercellular mediators of inflammation.

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High mobility group box-1 protein induces the migration and activation of human dendritic cells and acts as an alarmin.

Yang D, Chen Q, Yang H, Tracey KJ, Bustin M, Oppenheim JJ.

J Leukoc Biol. 2007 Jan;81(1):59-66. Epub 2006 Sep 11.

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The relationship between apoptosis and high-mobility group protein 1 release from murine macrophages stimulated with lipopolysaccharide or polyinosinic-polycytidylic acid.

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HMGB1 as a DNA-binding cytokine.

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Release of high mobility group box 1 by dendritic cells controls T cell activation via the receptor for advanced glycation end products.

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HMGB1 expression and release by bone cells.

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[Role of HMGB1 in rheumatic diseases]

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High mobility group 1 B-box mediates activation of human endothelium.

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RAGE is the major receptor for the proinflammatory activity of HMGB1 in rodent macrophages.

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HMGB1 in sepsis.

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