Autophagy induction and CHOP under-expression promotes survival of fibroblasts from rheumatoid arthritis patients under endoplasmic reticulum stress
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* Corresponding author: Han-Jung Chae hjchae@chonbuk.ac.kr
1 Department of Rheumatology, Medical School, the Catholic University of Korea, Seoul, Republic of Korea, 150-713
2 Department of Pharmacology and Cardiovascular Research Institute, Medical School, Chonbuk University, Jeonju, Chonbuk, Republic of Korea, 561-181
3 Division of Rheumatology, Department of Internal Medicine, Medical School, Chonbuk University, Jeonju, Chonbuk, Republic of Korea, 561-181
4 Division of Rheumatology, Department of Internal Medicine, Kyungpook University Hospital, Daegu, Republic of Korea, 561-181
5 Department of Biochemistry and Cell Biology, School of Medicine, Kyungpook University, Daegu, Republic of Korea, 110-749
6 Department of Internal Medicine, Medical School, Chonbuk Univ, Jeonju, Republic of Korea, 561-181
7 Research Center for Pulmonary Disorders, Chonbuk Hospital, Jeonju, Republic of Korea, 561-181
8 Department of Neurology, Medical School, Chonbuk University, Jeonju, Chonbuk, Republic of Korea, 561-181
9 Department of Anatomy, Medical School, Chonbuk University, Jeonju, Chonbuk, Republic of Korea, 561-181
10 Biochip Research Center, Hoseo University, Chungnam, Republic of Korea, 336-795
11 Department of Dental Pharmacology, Dental School, Wonkwang University, Iksan, Chonbuk, Republic of Korea, 570-749
Arthritis Research & Therapy 2010, 12:R19 doi:10.1186/ar2921
Published: 1 February 2010Additional files
Additional file 1:
Electron microscopy data. Electron microscopy data from thapsigargin (1 μM)-treated OASF and RASF.
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