Table 2

Factors affecting susceptibility/resistance to adjuvant arthritis

Effector pathways/response
Susceptibility/resistance (+/-)
Rat strains tested
References

MHC and non-MHC genes including particular quantitative trait loci influence arthritis susceptibility
+/-
Table 1
Table 1, [4,5]
Differential T-cell proliferative and/or cytokine response to Bhsp65, its arthritogenic epitope 180 to 188, or its regulatory C-terminal determinants
+/-
LEW/WKY/Wistar/F344
[20,24,30,31,33,36]
Increased T-cell response to Rhsp65 and its regulatory epitopes
-
LEW/WKY
[20,24,26]
Anti-Bhsp65/Rhsp65 antibody response induced upon Mtb challenge
-
LEW/BN/WKY/F344/Wistar
[23,25,43]
Increased expression of hsp47 in the joints and enhanced anti-hsp47 antibodies in rats with arthritis
+
DA/AO
[45]
Immune response to hsp71
-
DA/AO/LEW
[45,46]
Migration into and retention within the target organ (joints) of arthritogenic leukocytes; the role of monocyte chemoattractant protein 1 and monocyte/macrophage chemotaxis
+
LEW/WKY
[19,20]
Increased reactive oxygen species contributing to arthritis resistance
-
DA/AO
[10]
Blunted hypothalamic–pituitary–adrenal axis activity
+
LEW/F344/Wistar
[51,53-55]
Microbial flora in a conventional housing environment
-
F344/Wistar
[56,59,60]

Bhsp65, mycobacterial heat-shock protein 65; hsp, heat-shock protein; MHC, major histocompatibility complex; Mtb, Mycobacterium tuberculosis H37Ra; Rhsp65, rat heat-shock protein 65.

Kim and Moudgil Arthritis Research & Therapy 2009 11:239   doi:10.1186/ar2755