A prospective study of androgen levels, hormone-related genes and risk of rheumatoid arthritis
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* Corresponding author: Elizabeth W Karlson ekarlson@partners.org
1 Division of Rheumatology, Immunology, and Allergy, Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, 75 Francis Street, Boston, MA 02115, USA
2 Channing Laboratory, Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, 75 Francis Street, Boston, MA 02115, USA
3 Department of Epidemiology, Harvard School of Public Health, 677 Huntington Avenue, Boston, MA 02115, USA
4 Immune Disease Institute, 800 Huntington Avenue, Boston, MA 02115, USA
5 Genzyme Corporation, 500 Kendall Street, Cambridge, MA 02115, USA
6 Division of Preventive Medicine, Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, 75 Francis Street, Boston, MA 02115, USA
7 Division of Aging, Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, 75 Francis Street, Boston, MA 02115, USA
8 Department of Ambulatory Care and Prevention, Harvard Medical School, 25 Shattuck Street, Boston, MA 02115, USA
Arthritis Research & Therapy 2009, 11:R97 doi:10.1186/ar2742
See related editorial by Cutolo, http://arthritis-research.com/content/11/5/126
Published: 25 June 2009Additional files
Additional file 1:
A Word file containing two tables that list the association of htSNPs in the AR gene and RA. Table S1 presents the association of the six htSNPs in the AR gene with RA in the NHS, in the WHS, and in the pooled sample. Table S2 presents the association of the six htSNPs in the AR gene with seropositive RA and seronegative RA in the NHS, in the WHS, and in the pooled sample.
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